松阪 泰二

Matsusaka Taiji

  • 教授
  • 学位:博士(医学)

基本情報

所属

  • Undergraduate School of Medicine / Faculty of Medicine
  • Graduate School of Medicine / Course of Advanced Medical Science
  • Graduate School of Medicine / Course of Medical Science

研究と関連するSDGs

  • Good Health and Well-Being

詳細情報

研究キーワード

  • proteinuria
  • kidney diseases
  • podocyte
  • Transgenic mouse
  • Renin-angiotensin
  • Cardio vascular
  • Kidney

研究分野

  • Life sciences Nephrology

受賞

  • 1997 Young Investigator Grand of National Kidney Foundation
  • 1995 National Kidney Foundation Fellowship

論文

Dual deletion of guanylyl cyclase-A and p38 mitogen-activated protein kinase in podocytes with aldosterone administration causes glomerular intra-capillary thrombi.

TFEB-mediated lysosomal exocytosis alleviates high fat diet-induced lipotoxicity in the kidney.

Podocytes are lost from glomeruli before completing apoptosis.

Kidney Angiotensin in Cardiovascular Disease: Formation and Drug Targeting.

講演?口頭発表等

  • マウスを用いた腎臓病の研究

所属学会

  • International Society of Nephrology
  • Japanese Society of Nephrology
  • American Society of Nephrology

共同研究?競争的資金等の研究課題

Inflammation-like reactions within podocytes caused by mitochondrial stress

Roles of Foxc1/2 in nephron development

High-throughput verification of pathways regulating kidney diseases

Next generation therapy of podocytopathy developed by renal organoid

Podocyte-tubular epithelial cell interaction plays a role to prevent tubular injury

Screening of novel podocyte protective reagents using nephrin reporter mice

Integration of proteomics and metabolomics for drug discovery based on a search for the pathophysiology in glomerulosclerosis

Foxc1/2 transcription factors and glomerulosclerosis

Effect of genetic inactivation of Keap1 on the kidney morphogenesis in mice

a trial for generation of parietal podocytes

Novel Approach for Treatment of Glomeruloclerosis

Mechanisms of progression of chronic kidney disease

Involvement of antioxidant defense mechanisms in the pathogenesis of renal damage

Roles of Foxc transcription factors in podocyte injury.

Mechanism of podocyte injury

Recovery from Glomerular Sclerosis

Development of congenital anomalies of the kidney and urinary tract (CAKUT)

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